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with 100% for the control ( Meyer and Hausbeck, 2013 ). Despite the effectiveness of soil-applied fungicides in phytophthora management, the labels of oomycete-specific fungicides may not support this application method ( Bird et al., 2016 ; Sanogo and

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, caused by the oomycete pathogen Phytophthora capsici , is a severe disease affecting squash and pumpkin. Originally described in 1922 after being identified on pepper [ Capsicum annum ( Leonian, 1922 )], it was first reported on cucurbits (Cucurbitaceae

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Kim, 1995 ; Oelke et al., 2003 ). Phytophthora blight is caused by the oomycete pathogen Phytophthora capsici, which can infect a wide range of vegetable crops ( Crossan et al., 1954 ; Polach and Webster, 1972 ). Management of phytophthora blight

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disease is caused by the oomycete pathogen Pseudoperonospora cubensis (Berk. & Curt.) Rostov., which has a host range consisting of more than 60 species belonging to 20 genera in the Cucurbitaceae family, and includes important crops such as cucumber

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of seedlings to oomycete pathogens ( Koh et al., 1987 ; Lazarovits et al., 1981 ; McClure and Robbins, 1942 ; Mellano et al., 1970 ). Vegetable crops in the Cucurbitaceae and Solanaceae families develop ARR to P. capsici fruit rot ( Ando et al

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has specificity toward oomycetes ( Lyr, 1995 ; Vuik et al., 1990 ). Etridiazole disrupts the lipid structure of cell membranes ( Radzuhn and Lyr, 1984 ) and inhibits respiration by binding to the mitochondrial membrane between cytochromes b and c

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The oomycete Phytophthora phaseoli is one of the most threatening pathogens of lima bean (Phaseolus lunatus) in the humid Mid-Atlantic United States. In the last 60 years, P. phaseoli has evolved to overcome genetic resistance in the host and several physiological races have been identified during the last 6 decades. Six physiological races A, B, C, D, E, and F have been identified over the years. Only race F has been detected in the field over the past decade. Identifying and characterizing sources of resistance to this pathogen and determining the nature of resistance were the main objectives. Eight commercial cultivars and 35 germplasm accessions of P. lunatus were evaluated for their reaction to races E and F. Four commercial cultivars and four accessions with resistance to race E, and two cultivars and four accessions with resistance to race F were identified. None of the germplasm evaluated were resistant to both races. Five populations of F2 plants and a recombinant inbred line (RIL) population were produced and inoculated to investigate the inheritance of resistance to races E and F. Resistance to races E and F was determined to be conferred by single, independent, dominant genes.

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The oomycete plant pathogen Phytophthora capsici Leonian affects the cucurbit industry annually, in some cases causing 90% to 100% crop loss ( Babadoost, 2000 ; Meyer and Hausbeck, 2012 ). Michigan is a leading producer of processing squash

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economic impact P. capsici syndromes can have on cucurbit production. This oomycete pathogen affects a wide range of solanaceous and cucurbitaceous plants worldwide ( Erwin and Ribeiro, 1996 ; Tian and Babadoost, 2004 ). Infection can occur at any plant

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pepper varieties with tolerance to the disease phytophthora blight, whereas it is less common on more susceptible varieties ( Kline et al., 2011 ). Phytophthora blight is a soilborne disease caused by the oomycete Phytophthora capsici ( Leonian, 1922

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