in the susceptibility of these dedicated pollenizers to fusarium wilt, caused by the soilborne fungus FON, in commercial fields. Pollenizers susceptible to fusarium wilt appear to produce fewer staminate flowers making them less effective as
–vermiculite formulation based on T. harzianum (CECT 20714) and the use of this agent as a nonformulated conidia suspension (direct inoculation) were both tested for their potential use as inocula for controlling fusarium wilt and promoting melon plant growth under in
or as a saprophyte in vegetal residues ( Louws et al., 2010 ). Chemical control of fusarium wilt is limited by the number of chemicals available and their effectiveness ( Martyn, 2012 ), and increasing concern about agriculture sustainability
Oriental melon ( Cucumis melo var. makuwa Makino) is an important germplasm resource of melon in China and is cultivated in Fuzhou (Fujian Province, China) for its delicious sweetness, high nutrient quality, and flavor. Fusarium wilt of Oriental
Three races of Fusarium oxysporum f.sp. lactucae, cause of fusarium wilt of lettuce, are known in Japan, where the pathogen was first observed in 1955. Fusarium wilt first affected commercial U.S. lettuce production in 1990 in Huron, Calif., but did not become a serious problem in the U.S. until 2001 when it reappeared in Huron and appeared in the Yuma, Arizona lettuce production area. Reactions of three fusarium wilt differentials (`Patriot', susceptible to races 1, 2 and 3; `Costa Rica No. 4', resistant to race 1, and susceptible to races 2 and 3; and `Banchu Red Fire', susceptible to races 1 and 3, and resistant to race 2) in a naturally-infected commercial field test and artificially-inoculated greenhouse tests, indicated presence of race 1 in the Yuma lettuce production area. Reactions of these differentials to an isolate from Huron confirmed the presence of race 1 in that area. Consistent with previous results from the U.S. and Japan, `Salinas' and `Salinas 88' were resistant to the Yuma and Huron isolates of race 1, whereas `Vanguard' was highly susceptible. Limited F1 and F2 data indicate that resistance to race 1 in `Costa Rica No. 4' and `Salinas' is recessive. `Calmar' is the likely source of resistance in `Salinas' and `Salinas 88'.
Cucumber ( Cucumis sativus L.) is one of the major greenhouse vegetables in the world and is very vulnerable to fusarium wilt caused by Fusarium oxysporum (FO) ( Ahn et al., 1997 ; Ye et al., 2004 ). Fusarium pathogen infects the roots and
Tomato (Lvcopersicon esculentum) line E427 contains Fusarium wilt resistant gene I-3 on chromosome 7 and I-2 (and presumably the linked I) genes on chromosome 11. E427 was crossed with `Bonny Best' (i, i-2, i-3) and backcrosses (BC) to `Bonny Best' and F2, seed were obtained. Self pollination of 187 BC and 150 F2 plants were made. Progeny were screened against Fusarium races 1, 2, and 3 and lines with recombinant ratios were self-pollinated and rescreened until homozygous. Five lines were resistant to races 2 and 3 but susceptible to race 1. These had the isozyme band got-2 linked to I-3, RFLP markers linked to I-3 and no RFLP markers linked to I-2. Five lines were resistant to race 1 but susceptible to races 2 and 3. These had the susceptible qot-2 band and no RFLP markers linked to I-3 or I-2. F2 complementation tests of 2 of these lines with `Manapal' (I) indicated they contained I. Three lines were resistant to race 2 but susceptible to races 1 and 3. These had the susceptible qot-2 band, I-2 linked RFLP markers, and no I-3 linked RFLP markers.
Abstract
U.C. PMR 45 fusarium wilt-resistant (Foml) is an andromonoecious, salmon-orange flesh, western shipping-type muskmelon, Cucumis melo L. It has value as a germplasm source for selection as well as for the development of commercial hybrids.
Abstract
We obtained estimates of genetic components of variance for resistance to Fusarium wilt in sweet-potato, through study of 40 randomly chosen plants of an intermating population and their respective open-pollinated offspring. The additive component accounted for virtually all of the genetic variance. Predicted advance from mass selection of the best 10% of the parents was 10.5% of the mean. The actual advance obtained in offspring of the selected plants was 13.9%. Heritability was estimated as .86.
Abstract
The mode of inheritance of resistance to Fusarium wilt (Fusarium oxysporum f. sp. melonis) races 0 and 2 in muskmelon (Cucumis melo L.) cv Perlita FR and race 2 in ‘Doublon’ was determined by analyzing segregation of F1, F2, F3, and BC1 populations of crosses with susceptible ‘PMR 45’. The ratios obtained indicate that resistance to both races 0 and 2 in ‘Perlita FR’ is conferred by a dominant gene. The segregation data indicate that resistance to race 2 in ‘Doublon’ also is conferred by a dominant gene. Allelism tests indicated that gene Fom-1, which controls resistance to race 0 in ‘Doublon’, is different from the gene controlling resistance to race 0 in ‘Perlita FR’ and that 2 different genes confer resistance to race 2 in ‘Doublon’ and ‘Perlita FR’. The reaction of ‘Perlita FR’ and ‘Doublon’ backcross progenies of (resistant × susceptible) × susceptible, inoculated simultaneously or sequentially with race 0 and race 2, suggest that gene Fom-1 confers resistance to races 0 and 2 in ‘Doublon’ and that a single gene confers resistance to both races 0 and 2 in ‘Perlita FR’. The resistant dominant gene in ‘Perlita FR’ is designated F. oxysporum f. sp. melonis 3 (Fom-3). The source of Fusarium wilt resistance in ‘Perlita FR’ is discussed. ‘Perlita FR’ was found susceptible to race 1 and race 1, 2.