; Gordillo et al., 2008 ). Both genes, Sw-5 for resistance to TSWV and Ph-3 for resistance to P. infestan s, were originally introgressed from wild relatives and confer high levels of pathogen resistance. The TSWV resistance gene Sw-5 was
receive the soil topdressing treatment. The plants with N treatment were irrigated with quarter-strength N-free Hoagland’s solution ( Hoagland and Arnon, 1950 ) with 2 m m NH 4 NO 3 at pH 6.5; those without N treatment were irrigated with the same
the contents of Fe and Al, which were much higher in Pd than in Pl, the other metal elements did not show differences between the Pl and Pd petals. Table 3. Concentration of metal elements in Paeonia delavayi petals. Besides, the pH of the Pl and Pd
Flower color results from the interaction of a pigment (anthocyanin) with a co-pigment (usually a flavonone or flavonol) at a specific pH. At more alkaline pHs (pH 5 to 6), an anthocyanin/co-pigment complex is blue; while at more acidic pHs (pH 3-4), the same anthocyanin/copigment complex is red. In Phalaenopsis pulcherrima, a mutation in pH resulted in a bluer flower color. The difference in pH between the normal-colored magenta flowers (pH 5.8) and mutant violet flowers (pH 5.5) was due to a single co-dominantly inherited gene.
‘Mountain Bebe’ is the F 1 hybrid of NC 7 Grape × NC 8 Grape. It is resistant to late blight ( Ph-2 and Ph-3 genes), tomato spotted wilt virus ( Sw-5 gene), and fusarium wilt races 2 and 3 ( I-2 and I-3 genes). The hybrid has a compact
1 Current address: Department of Horticulture, University of Arkansas, Fayetteville, AR 72701. 2 Current address: Department of Horticulture and Landscape Architecture, Colorado State University, Fort Collins, CO 80523-1173. 3 To whom reprint
oxysporum f.sp. lycopersici (Sacc.) Snyd. and Hans.) (races 1 and 2) ( I/I and I-2/I-2 genes), late blight (LB) ( Ph-2/ph-2 and Ph-3/ph-3 genes), Tomato mosaic virus (ToMV) ( Tm2/tm2 gene), and Tomato spotted wilt virus (TSWV) ( Sw-5/sw-5 gene
03220 (x)-20, a large-fruited, early blight/late blight-resistant tomato line having the Ph-2 and Ph-3 genes combined for late blight resistance ( Fig. 1 ). The resultant 0463 hybrid was then crossed with 9722 (x)-18, an early blight/late blight
Abstract
Organoleptic tests of the non-ripening tomato mutants rin and nor and their F1 hybrids with the normal-fruit-bearing cultivar ‘Rutgers’ indicated that fruits of the rin heterozygous plants (rin/+) were slightly inferior and that those of inferior in flavor to fruits of the normal genotype (+/+), all sampled 3–5 days after ethylene and CO2 evolution rates attained maximum levels. The flavor of fruits of the double heterozygote nor heterozygotes (nor/+) were distinctly rin/+, nor/+ was poorer than either of the 2 single-gene heterozygotes, while fruits of both homozygous plants, nor/nor and rin/rin, were unpalatable. Analyses of pH, titratable acidity, total soluble solids, and reducing sugars did not indicate that any of these parameters is responsible for the inferior flavor of the genotypes containing the non-ripening genes. Comparisons of reciprocal crosses provided no evidence of cytoplasmic inheritance of fruit flavor.
Abstract
The abstract for the paper, “Effect of the Fruit-ripening Mutant Genes rin and nor on the Flavor of Tomato Fruit” by E. Kopeliovitch, Y. Mizrahi, H. D. Rabinowitch, and N. Kedar [J. Amer. Soc. Hort. Sci. 107(3):361–364. 1982.], contains several errors. The correct version of the abstract is as follows: Organoleptic tests of the nonripening tomato mutants rin and nor and their F1 hybrids with the normal-fruit-bearing cultivar ‘Rutgers’ indicated that fruits of the rin heterozygous plants (rin/+) were slightly inferior and that those of heterozygote nor (nor/+) were distinctly inferior in flavor to fruits of the normal genotype (+/+), all sampled 3–5 days after ethylene and CO2 evolution rates attained maximum levels. The flavor of fruits of the double heterozygotes rin/+, nor/+ was poorer than either of the 2 single-gene heterozygotes, while fruits of both homozygous plants, nor/nor and rin/rin, were unpalatable. Analyses of pH, titratable acidity, total soluble solids, and reducing sugars did not indicate that any of these parameters are responsible for the inferior flavor of the genotypes containing the nonripening genes. Comparisons of reciprocal crosses provided no evidence of cytoplasmic inheritance of fruit flavor.