Tall fescue (Festuca arundinacea) and hybrid bluegrass (Poa pratensis L. × Poa arachnifera) can both be successfully grown in the transition zone of the United States. However, each grass has limitations. Tall fescue is susceptible to the fungal pathogen Rhizoctonia solani, whereas slow establishment and susceptibility to weed infestations limit hybrid bluegrass. Previous studies have shown the benefits of combining kentucky bluegrass with tall fescue in seeding mixtures. Research was conducted to evaluate the impact of two seeding combinations of hybrid bluegrass and tall fescue (one combination seeded at a 1.9:1 seed count ratio favoring tall fescue, the other combination seeded at a 1:1.8 seed count ratio favoring hybrid bluegrass) as well as monocultures of the species on turfgrass cover, weed species infestation, brown patch disease severity caused by R. solani, sod strength and species ecology. The seeding combinations had lower weed density during establishment and greater turf cover than the monoculture of hybrid bluegrass. The monoculture of tall fescue was subjected to more brown patch disease than the seeding combinations during and after the first year of establishment. Brown patch infestations likely reduced tall fescue cover and led to a species shift favoring hybrid bluegrass in the seeding combinations based on tiller count and weight data. Seeding combinations of tall fescue and hybrid bluegrass are beneficial from an epidemiological perspective because they reduce disease and weed infestations compared with monocultures of either species. From an agronomic perspective, the seeding combination favoring tall fescue provided the densest turf, whereas the seeding combination favoring hybrid had the greatest sod strength. Chemical name used: clopyralid (3,6 dichloropyridine-2 carboxylic acid)
Matthew A. Cutulle, Jeffrey F. Derr, David McCall, Brandon Horvath, and Adam D. Nichols
Patrick A. Jones, James T. Brosnan, Gregory K. Breeden, José J. Vargas, Brandon J. Horvath, and John C. Sorochan
Divoting is a common occurrence on golf courses and athletic fields. Research was conducted at the University of Tennessee Center for Athletic Field Safety (Knoxville, TN) during 2012–13 evaluating the effects of preemergence (PRE) herbicide applications on hybrid bermudagrass [C. dactylon (L.) Pers. × C. transvaalensis Burtt-Davy, cv. Tifway] divot resistance and recovery. Plots were subjected to the factorial combination of seven herbicide treatments (indaziflam at 35 and 52.5 g·ha−1; prodiamine at 840 g·ha−1; pendimethalin at 3360 g·ha−1; dithiopyr at 560 g·ha−1; oxadiazon at 3360 g·ha−1; non-treated control) and three divot timings [1, 2, and 3 months after herbicide treatment (MAT)]. Rates were based on label recommendations for preemergence crabgrass (Digitaria spp.) control. Herbicides were applied on 15 Mar. 2012 and 2013. Divots were generated using a weighted pendulum apparatus designed to impart 531 J of impact energy to the turf sward with a golf club. Divot resistance was quantified by measuring divot volume at each timing while divot recovery was quantified by measuring turf cover in the divot scar using digital image analysis. All herbicide-treated plots produced divots with volumes ≤ the non-treated control. In 2013, volumes were greater for divots produced 1 MAT (215 cm3) than those created 2 MAT (191 cm3) or 3 MAT (157 cm3). No differences in divot recovery were detected as a result of herbicide treatment in either year. Under the conditions of this study, applications of PRE herbicides at labeled rates did not affect divot resistance or recovery.
Chemical names: N-[(1R,2S)-2,3-dihydro-2,6-dimethyl-1H-inden-1-yl]-6-(1-fluoroethyl)-1,3,5-triazine-2,4-diamine (indaziflam), 2,4 dinitro-N3,N3-dipropyl-6-(trifluoromethyl)-1,3-benzenediamine (prodiamine), N-(1-ethylpropyl)-3,4-dimethyl-2,6-dinitrobenzenamine (pendimethalin), S,S-dimethyl 2-(difluoromethyl)-4-(2-methylpropyl)-6-(trifluoromethyl)-3,5-pyridinedicarbothioate (dithiopyr), 3-[2,4-dichloro-5-(1-methylethoxy)phenyl]-5-(1,1-dimethylethyl)-1,3,4-oxadiazol-2-(3H)-one (oxadiazon)