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David T. Handley

Frost injury on strawberry flowers (cv. Redchief) was studied in a field over two seasons. During the first season, a light frost during anthesis caused varying degrees of injury to blossoms. Five grades were assigned to the blossoms according to the degree of injury observed. The resulting fruit malformations correlated to the severity of blossom injury, ranging from no development (blossom death) when flower receptacles were completely black, to slight dimpling when only a portion of the receptacle had been discolored. During the second season, a colder frost occurring at the bud stage caused generally greater injury to the blossoms. The range of injury was less variable, therefore only three grades were assigned to the blossoms. The resulting fruit malformation again related to the intensity of blossom injury. Frost has long been understood to kill unprotected strawberry blossoms. This study has shown that nonfatal frost injury to strawberry blossoms can result in a variety of fruit malformations which previously may have been attributed to other causes.

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Hanan M. El-Hoseiny, Mohamed N. Helaly, Nabil I. Elsheery, and Shamel M. Alam-Eldein

off-year ( El-Motaium et al., 2019 ). Under salinity stress conditions, the application of ZnO and Si nanoparticles has improved plant resistance to salinity, reduced floral malformation percentage, minimized the incidence of alternate bearing, and

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David T. Handley and James E. Pollard

Greenhouse experiments were designed to study conditions affecting strawberry malformation caused by the tarnished plant bug (TPB). Duration of blossom exposure to TPB affected the type of malformation. Exposure at anthesis for 8 hours caused visible deformity. Exposure for 48 hours caused some apical seediness, the malformation most commonly associated with TPB. Continuous exposure to TPB usually caused blossom death. Increased exposure to TPB caused a higher percentage of nonviable achenes per strawberry. Some effects appeared to be cultivar-dependent. Honeoye strawberries were less likely to show apical seediness than Redchief strawberries, but were more likely to experience blossom death. Malformation was also affected by strawberry development stage at the time of TPB feeding. Feeding at prebloom caused blossom death. Feeding at petal fall or achene seperation resulted in fruit malformation, about half of which was apical seediness. Feeding at pink receptacle stage caused little visible damage.

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Clinton J. Hunnicutt, Andrew W. MacRae, and Vance M. Whitaker

, Cary, NC) to investigate the effect of herbicide rate and strawberry cultivar on new leaf production, percentage of malformed leaves, and marketable yield. The leaf malformation data are presented as the percentage of new leaves that become malformed

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Michele A. Stanton, Joseph C. Scheerens, Richard C. Funt, and John R. Clark

malformation, and flowers that did not abscise; a subsequent reduction in yield was observed. Male floral competence of LT- and MT-grown plants. The majority of flowers produced at the two lower temperature regimens (LT and MT) had normal

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Rebecca L. Darnell, Nicacio Cruz-Huerta, and Jeffrey G. Williamson

. Moshkovits, H. Rosenfeld, K. Shaked, R. Cohen, M. Aloni, B. Pressman, E. 1998 Varietal differences in the susceptibility to pointed fruit malformation in tomatoes: Histological studies of the ovaries Sci. Hort. 77 145 154 Van-de-Dijk, S.J. Maris, J.A. 1985

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David T. Handley and James E. Pollard

The tarnished plant bug (Lvgus lineolaris) is a serious pest of strawberries in North America, causing a severe malformation of the receptacle known as “apical seediness” or “buttoning”. Light and scanning electron microscopy were used to assess tarnished plant bug feeding on strawberries and to determine the nature of the injury. During early fruit development stages (anthesis to petal fall) the primary feeding sites were developing achenes. Feeding sites on more developed fruit changed to receptacle tissue, usually close to an achene. The “buttoning” malformation of strawberries associated with tarnished plant bug is most likely a result of the destruction of achenes during early fruit development stages. Feeding on receptacle tissue later in fruit development causes more localized damage, such as creases and indentations.

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Karsedis Distabanjong and Robert L. Geneve

Somatic embryos from Eastern redbud show a high degree of malformation during development and a low conversion rate to seedlings. This problem is common with somatic embryo systems, especially with legume species. A procedure for multiple shoot formation from somatic embryo explants of Eastern redbud was developed that bypasses the need for germination to recover plantlets. Somatic embryo explants cultured on DKW medium containing benzyladenine (BA) and thidiazuron (TDZ) produced more shoots than either treatment alone. The highest number of shoots (3.3 to 3.4 shoots per explant) was obtained from partially desiccated and wounded explants treated with a combination of 5 or 10 M BA and 0.5 or 1.0 M TDZ for 20 days before being transferred to the same medium without TDZ. The number of shoots formed was increased from 1.5 to 3.2 shoots per explant by cutting through the cotyledonary node prior to culture. In addition, the frequency of explants forming shoots was increased by desiccation of somatic embryo explants to ≈50% moisture and by using somatic embryos with two well-formed cotyledons as explants.

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R. Nunez-Elisea, M. L. Caldeira, and T. L. Davenport

Thidiazuron (TDZ; N-phenyl-N-1,2,3-thiadiazol-5-ylurea) stimulates axillary bud break in some horticultural crops. We are exploring its ability to initiate bud growth in mango trees in order to manipulate vegetative and reproductive shoot initiation. Axillary buds on defoliated, decapitated shoots were treated in late October, 1989 (about two months before normal floral initiation), with 0, 125, or 1000 ppm TDZ. Although timing or percent of bud-break was unaffected by TDZ, the compound influenced growth expression. TDZ (125 ppm) produced morphologically typical panicles (mixed or purely floral), while at 1000 ppm purely floral panicles were produced which were abnormally compact (similar to panicles affected by mango malformation). Non-treated buds produced only vegetative shoots. Sprays of TDZ (25 to 200 ppm) on developing panicles produced morphological anomalies in panicles such as thickening of the central axis and secondary branches, increase in flower size, and sprouting of the most basal buds on the central axis. Effect during the vegetative flushing period will be discussed.

Free access

R. Nunez-Elisea, M. L. Caldeira, and T. L. Davenport

Thidiazuron (TDZ; N-phenyl-N-1,2,3-thiadiazol-5-ylurea) stimulates axillary bud break in some horticultural crops. We are exploring its ability to initiate bud growth in mango trees in order to manipulate vegetative and reproductive shoot initiation. Axillary buds on defoliated, decapitated shoots were treated in late October, 1989 (about two months before normal floral initiation), with 0, 125, or 1000 ppm TDZ. Although timing or percent of bud-break was unaffected by TDZ, the compound influenced growth expression. TDZ (125 ppm) produced morphologically typical panicles (mixed or purely floral), while at 1000 ppm purely floral panicles were produced which were abnormally compact (similar to panicles affected by mango malformation). Non-treated buds produced only vegetative shoots. Sprays of TDZ (25 to 200 ppm) on developing panicles produced morphological anomalies in panicles such as thickening of the central axis and secondary branches, increase in flower size, and sprouting of the most basal buds on the central axis. Effect during the vegetative flushing period will be discussed.