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Matthew D. Taylor, Paul V. Nelson, and Jonathan M. Frantz

spots on young leaves; dark green color, large purplish or black spots, leaf cupping, and interveinal, marginal, or speckled chlorosis on the entire plant; cupping, and interveinal, marginal, or speckled chlorosis or necrosis on older leaves; and in

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Jonathan M. Frantz, Sushant Khandekar, and Scott Leisner

stunted and displayed interveinal chlorosis reminiscent of Fe deficiency ( Fig. 1A–D ). Bucher and Schenk (2000) reported that Cu toxicity symptoms can be alleviated, at least temporarily, with Fe additions, which would certainly confuse the diagnosis of

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Chad T. Miller, Neil S. Mattson, and William B. Miller

rating scale from 0 to 5: 0 = no chlorosis; 1 = minimal chlorosis, slight yellowing; 2 = general leaf yellowing; 3 = progressed leaf yellowing with initial stages of green veins; 4 = distinct interveinal chlorosis; and 5 = severe chlorosis, often

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Mary Jane Clark and Youbin Zheng

mS·cm −1 = 1 mmho/cm and 1 ppm = 1 mg·kg −1 . Nutrient deficiency symptoms. Interveinal chlorosis was observed for all three species and developed on young petunia leaves by 5 WAT for MIX 1 and MIX 5 and by 10 WAT for MIX 2 and MIX 7. In addition

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Axel O. Ramírez-Madera and Michael J. Havey

. Kabelka et al. (1997) found that inoculating cotyledons of Dina-1 resulted in interveinal chlorosis on lower leaves; whereas TMG-1 had no symptoms regardless of inoculant placement. Svoboda et al. (2013) reported A192-18, G22, and TMG-1 as immune to

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Concetta Licciardello, Biagio Torrisi, Maria Allegra, Fabiola Sciacca, Giancarlo Roccuzzo, Francesco Intrigliolo, Giuseppe Reforgiato Recupero, Paola Tononi, Massimo Delledonne, and Vera Muccilli

availability ( Mengel, 1994 ) and the development of Fe chlorosis symptoms. The most common Fe chlorosis symptoms include interveinal chlorosis in young leaves ( Abadía and Abadía, 1993 ; Morales et al., 1998 ), decreased leaf net photosynthetic rate ( Briat

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Scovia Adikini, Settumba B. Mukasa, Robert O.M. Mwanga, and Richard W. Gibson

viruses using nitrocellulose membrane–enzyme-linked immunosorbent assay (NCM-ELISA). Fig. 2. Sweetpotato root sprout of ‘NASPOT 1’ showing deviation of leaf morphology from normal. ( A ) ‘NASPOT 1’ with interveinal chlorosis and distorted leaf shape. ( B

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Robert M. Pyne, Adolfina R. Koroch, Christian A. Wyenandt, and James E. Simon

et al., 2006 ; Nagy and Horvath, 2011 ). The initial disease symptom is usually interveinal chlorosis observed on the adaxial leaf surface, which can be ambiguous and easily mistaken for nutrient deficiency allowing the pathogen to persist under the

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Timothy K. Broschat

as an interveinal chlorosis of these same-aged leaves ( Broschat, 2008 ; Dickey, 1983 ). Some trees display deficiency symptoms of both elements simultaneously, but typically only one element is deficient on a particular tree at any point in time

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Joseph P. Albano, James Altland, Donald J. Merhaut, Sandra B. Wilson, and P. Chris Wilson

(27.4467°N, 80.3256°W), that identified production problems related to high alkalinity was selected to be the source for plants, substrate, and water for the study. Affected plants at this nursery developed a general pattern of interveinal chlorosis on