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only affected the first flush of leaves. Fig. 3. ( A ) Leaf injury 15 d after treatment at the leaf unfolding stage with metolachlor at 1% (20 g·ha −1 ) of the field use rate. ( B ) The normal, noninjured second flush of leaves (bottom) with the

Mexican rootstocks. These correspond to the maximum values to avoid leaf injury and reduction in fruit yield. Other researchers have focused on Na + toxicity of tree crops rather than Cl – . For example, Na in the leaf tissue of tree crops in excess of 0

to six trees per soil sampling site. Table 2. Soil types, tree trunk sizes, leaflet sizes, leaf injury, and floor management practices used at the pecan test orchards. The orchards examined were planted with ‘Western Schley’ at various time periods

study as the lowest free chlorine concentration at which there was visible leaf injury and/or a significant reduction in any of the measured growth parameters. Visual injury (i.e., necrotic mottling, necrosis, chlorosis, premature abscission of foliage

studies. Solomakhin and Blanke (2010) reported a significant increase in leaf injury, because 10% to 42% of ‘Gala’ leaves and 15% to 32% of ‘Golden Delicious’ leaves were injured per limb with increased string thinning severity. However, leaves were only

traffic stress, although differences in LWC were found between japanese zoysiagrass and kentucky bluegrass under light and moderate stress levels. The LWC reduction associated with traffic stress may be attributed to leaf injury ( Beard, 2005 ). Traffic

injury data were collected on 12 Mar. 2012 and again on 17 Jan. 2014. Plants were visually evaluated for damage using a scale from 1 to 6 as follows: 1 = no injury; 2 = minor leaf injury; 3 = leaf/stem damage on outer 30%; 4 = leaf/stem injury on outer 60

back to the leaves and stem, hence resulting in the large drop in the sucrose concentration on Day 21 ( Fig. 3E ). Previous articles reported that leaf injury to Eustoma cut flowers treated with sucrose pulsing or vase solution is caused by excessive

artificially controlled CA regimes. In comparative analyses, freezing injury in W6 5018 initiated and reached the maximum earlier than Jemalong-6 in all CA regimes. Webb et al. (1994) demonstrated genotypic differences in leaf injury, rate of increase in

·L −1 . At the end of week 5, plants exposed to the highest concentrations of salinity showed severe leaf injury and general water stress symptoms ( Fig. 3 ). Leaf damage and adverse effects on growth were visibly evident on plants exposed to Na at 0.9 g